DREB-D1 .

Note: BW = CIMMYT wheat accession number. BDV2

7Ai#1L group.7DL = T7DS.7DL7Ai#1L {0182}, {552}.

7DL-7EL {10157}.

sb2 . [ Tsn1 {11255}.

QTL Yangmai 6 (R)/Sonalika (S): RIL population: AUDPC was controlled by four QTLs derived from Yangmai 6, viz. QSb.bhu-2AL ( Xbarc353-2A – Xgwm445-2A , R[2] =0.148), QSb.bhu-2BS ( Xgwm148-2B – Xgwm375-2B , R[2] =0.205), QSb.bhu-5BL ( Xgwm67-5BL – Xgwm371-5BL , R[2] =0.386) and QSb.bhu-6DL ( Xbarc173-6D – Xgwm732-6DL , R[2] =0.225) {10719}.

{939}; Festival {1554}; Fram I {130}; Huron CI 3315 {1175,1554}; Kenora {1554}; Kenya W744 = C6041 {1175,130}; Norka {1175,130}; Pika {130}; Sweden W1230 {1554}; Thew {1175}; TU 4 {130}; Zhengzhou 871124 {570}.

**PM2** TraesCS5D01G044600 {11503}.

[ Mlx {10885D {1007}; Pm48 {10935}. 5DS{945}.

Several alleles of Pm2 with wheat and alien origins have been reported in Chinese genotypes – see temporary designations. The complex nature of temporarily named powdery mildew resistance genes in the Pm2 region is discussed in {11380}. Several alleged alleles at the Pm2 locus are likely Pm2a {11503}.

1AS {947}, {943}.

The Pm3a, Pm3b, Pm3d and Pm3f alleles form a true allelic series based on sequence analysis {10292}. Following the cloning and sequencing of Pm3b {10064}, 6 other alleles were sequenced {10405}. The Chinese Spring (susceptible) allele, Pm3CS , considered to be ancestral and present in many hexaploid and tetraploid wheats, was also transcribed {10405, 10406}. Other wheats possessed a truncated sequence (e.g. Kavkaz), or were null {10405,10406}. Unique markers were developed for all 8 transcribed alleles, and for individual alleles {10405}. Alleles Pm3b, Pm3d , and Pm3f were detected in Scandinavian varieties using allele-specific markers {10681}.

TraesSYM2A03G00828360 {11774}.

2° {52}. 2AL {1464}.

A recessive resistance gene ( pmXXM {11661}) in Xiaomaomai had a similar protein structure to Pm4d , Pm4e and Pm4h {11661}. Pm4f . v: WATDE0571 {11775}. Pm4g . v: WW-740 {11775}. Pm4h . v: WW-474 {11774}. Pm4i . v: WATDE0048 {11775}. Pm4j . v: WATDE0592 {11775}. Add note at end of Pm4 section: Some variants of PM4 confer resistance to wheat blast {11632, 11735, 11775} – see Reaction to Magnaporthe grisea .

TraesCS7B02G441700 (susceptible allele) (chr7B: 706.811-706.816 Mb) {11533}.

Although Duanganmang ( PmDGM ) had an identical sequence and UTR to Pm5e , a second completely linked gene was postulated to account for a different response pattern to the Pm5e control {11705}. pm5.

C {626}; Abe {97,1256}; Coker747 {786, 1079}; Mengavi {97}; Oasis {786}; Timgalen {98}; TP 114/2[*] Starke deriv. B {626}.

4BL-5RL {543}, but more recently revised to T4BS.4BL-2R#IL {389}, {380}.

1R(1B).

6SL {572}, {598}. 6S[1] S {598}.

3BS-3S[1] #1S.

Pm14 and Pm15 were detected using hybrids between B. g. tritici and B. g. agropyri cultures.

Pm14 and Pm15 were detected using hybrids between B. g. tritici and B. g. agropyri cultures.

To account for the different chromosome locations a 4A-4B translocation was suggested {10217}. Based on the 5B location and similar disease responses Pm16 and Pm30 may be the same {10217}.

Deleted, see Pm1c .

6BL = T6BS.6R#2L {386}, {389}, {543}.

6VS#2 {1177, 11714}.

Deleted. Pm22 . Deleted, renamed as Pm1e

Deleted. Pm23 . Deleted, Renamed as Pm4c . PM24 . TraesCS1D02G058900; AET1Gv20142700. This locus is also named RMG1 ( RWT4 ) – see Reaction to Magniporthe grisea .

Linked with 3 RAPDs, the nearest, OPAG04950, at 12.8 +/- 4.0 cM {1343}; Associated with 3 RAPDs {1344}.

PM31 Deleted. This gene designation {0301} is not valid; subsequent studies {10918} showed the gene is Pm21 .

A further gene derived from T. monococcum PI 427772 was identified in BCBGT96A = PI 599036 = Saluda*3/PI 427772 {10479}. A single resistance gene was identified on chromosome 7AL in hexaploid germplasm NC96BGT4 (a T. monococcum derivative). This gene was proximal to Pm1 and considered to be different from Pm37 , although possibly allelic {10274}. **PM38** TraesCS7D03G0183600

triticina , Reaction to P. striiformis .

**bin:

PM53 Curator’s note: A publication of this gene could not be located.

5AS (5VS.5AL) {11108}. 5DS (5VS.5DL) {11109}.

5VS) {11109}.

6RS) {11155}.

Putative Pm65 allele {11752}. Pm351817 {11752}.

1VS#5) {11426}.

Pm8 was suppressed when locus Pm3 is transcribed (including Chinese Spring and Thatcher which have no currently detectable Pm3 resistance alleles) {10828}.

PMTR1 & **PMTR3I.** SECCE6Rv1G0382290 . PmTR1 &

6VS#4S {11703}.

Pm6S[l] conferred resistance to 28 of 30 Chinese Bgt isolates {11597}.

Recessive {10604}. 5A[m] L {10604}.

Three of 61 Israeli Bgt isolates were avirulent: all three isolates were from tetraploid wheat accessions. It is possible that the gene may be present in many common wheat accessions.

v: LS5082 {11629}. ma: Located in the interval 710.3 – 711.0 Mb {11629}.

TA2682c carries a second dominant gene located in chromosome 1A {10175}.

Unnnamed resistance gene {11612}. WTK4 {11612}. 7D {11612}.

Xbarc148-1A – Xgwmc550-1A interval. R[2] =0.074-0.099; QPm.caas-4DL proximal to Xwmc331-4D . R[2] =0.15-0.23; QPm.caas-6BS , proximal to Xbarc79-6BS , R[2] =0.09-0.13; and QPm.caas-7AL , proximal to Xbarc174-7AL , R[2] =0.067-0.071 {10680}. Additional temporarily named genes and QTL are listed in {11655}.

Susceptibility to this non-pathogen of common wheat is rare, with only one susceptible genotype being documented. A few susceptible tetraploid genotypes were identified {10939}.

VIGS silencing of 5AL-B4 on chromosome 5A compromised resistance conferred by Dn1 suggesting a decoy role {11333}. Tests of allelism indicated that Dn1, Dn2, Dn5, Dn6, and Dnx and four uncharacterized lines were identical or closely linked {11225}.

According to Saidi & Quick {1250}, Dn1 and Dn2 are probably allelic. Reference stocks with each gene showed allelism with a gene in PI 262605.

Dn4 and an uncharacterized gene in PI 151918 were allelic or tightly linked {11225}.

7DL {287}, {10396}, {10310}.

1B = 1BL.1RS {9918}.

QTL QTLs for antixenosis were associated with Xpsr687-7D (7DS) and Xgwm437-7D (7DL) in CS/CS (Synthetic 7D) {10136}. Separate antibiotic effects were demonstrated for the same chromosome {10136}. A QTL, QDn.unlp.6A , for antixenosis was associated with Xgwm1393-6AL and Xgwm1150-6AL in a CS/CS(Synthetic 6A) DH population {10216}.

The relationship of Fhb1 to Fhs1 or Fhsb2 {1096} is unknown. Lines combining Fhb1 and Sr2 are reported in {11170}; Fhb1 is located about 2 cM proximal to Sr2 . SYN1 / Ocoroni DH population: three QTL from SYN1 were identified, QFhs.cim-2D (PVE 25%), QFhs.cim-7A (PVE 4.7%) and Qfhs.cim-7A (PVE 4.2%) {11165}. A marker study found that 14 of 66 wheats with putative FHB resistance shared markers indicative of the 3BS QTL in Ning 7840, Sumai 3, Wangshuibai and possibly Wuhan 3, plus Japanese landraces Shinchunaga and Shirasu No 1 {10115}. The original source may be the landrace 'Taiwan Wheat' rather than Funo {10115}.

7Lr#1S{10529}.

Although plants with Fhb-4 were taller than the recurrent parent, the height difference was not associated with the Rht-B1 locus {10885}. Type I resistance (% infected plants) in this cross was attributed to 10 chromosome regions among which Qfhi.nau-4B ( Xwmc349-4B – Xgwm149-4B - r[2 ] = 0.75), XFhi.nau-5A (Xwmc96-5A – Xgwm304-5A - R[2 ] = 0.27) and Qfhi.nau-5B (Xgwm408-5B – Xbarc140-5B ) from Wangshuibai were detected in at least 3 of 4 years {10282}. A significant additive effect of QTL on 6D and 2A was also observed {10282}.

Closely linked in coupling with Qflw.nau-5A for narrow leaf width, but recombination is reported in {11041}. According to {11487} Fhb5 might be the same as Qfhs.ifa-5Ac but the issue remained ambivalent.

T1AL.1AS-1E[ts] #1S {11048}

T7DS.7D1-7e12L {11060}

QTL QTLs for resistance to Fusarium graminearum detected in the cross Renan/Recital {10069}. All resistance alleles, except QFhs.inra-3A , were contributed by Renan. LOD scores and percent of variation explained by the QT (R[2] ) are average of three years of field tests.

This QTL explained 42% of the variation in Sumai 3/Stoa {0175}. Two additional QTL for resistance to Fusarium graminearum were identified in the croSumai3/Stoa {0175}. The QTL on 4BS was associated with Xwg909-4B and the QTL on 6BS was associated with Xbarc101-6B and Xbcd1383-6B {0175}. The QTL associated with markers Xgwm493-3B/Xgwm533-3B (explaining 24.8 % of the variation), and Xbarc101-6B/Xbcd1383-6B were also identified in a RIL population from the cross ND2603/Butte 86 {0175}. In addition, one QTL on chromosome 3AL associated with Xbcd941-3A and one on chromosome 6AS associated with XksuH4-6A were identified in RILs from the cross ND2603/Butte 86 {0175}. Remus / CM-82036 (a Sumai 3 derivative): DH population: Resistance QTL on chromosome 3BS associated with Xgwm493-3B and Xgwm533-3B {0240}. Additional QTL in this cross were detected on chromosome 5A, associated with Xgwm293-5A and Xgwm304-5A , and possibly on 1B, associated with Glu-B1 {0240}. Two major genes with additive effects were reported in crosses between Sumai 3 (resistant) and two susceptible cultivars {0174}. One of the genes was assigned to 5AL based on linkage to the dominant awn suppressor B1 (RF 15.1-21.4%). Alve (S) / Line 685 R: DH population: QTL on chromosomes 4D ( Rht-D1 ), 3BS, 5A and 2BL {10972}. Two resistance QTL were needed to counteract the negative effect of the Rht-D1b semi-dwarfing allele {10972}. Arina (R) / Forno (S): Three QTLs, QFhs.fal-6DL (R[2] =22%), QFhs.fal-5BL.1 (in Forno, R[2] =14%) and QFhs.fal.4AL (R[2] =10%) and 5 minor QTLs in 2AL, 3AL, 3BL, 3DS and 5DL were detected {10172}. Arina / Riband DH lines: QTL affecting ADUPC were identified in 1BL(2), 2B, 4DS, 6BL and 7AL (Arina), and 7AL and 7BL (Riband). The most effective was the 4DS QTL that appeared to be an effect of Rht-D1a rather than height per se {10464}. Baishanyuehuang (R) / Jagger (S): RIL population: Four genes/QTLs derived from the resistant parent included Fhd1 (R[2] =0.16), Qfhb.hwwg-3BS c (R[2] =0.09), Qfhb.hwwg-3A (R[2] =0.05-0.08) and Qfhd.hwwg-5A (R[2] =0.05 in one trial) {10950}. Cansas (moderately resistant) / Ritmo (susceptible): Map based analysis across environments revealed seven QTL, QFhs.whs-1BS (1RS), QFhs.whs-3B (not Fhb1 ), QFhs.whs-3DL, QFhs.whs-5BL '(renamed Qfhs.lfl-1BL in {10768})' , QFhs.whs-7AL and QFhs.whs-7BL (cumultatively, R[2] = 0.56). The chromosome 1D gene was primarily involved in resistance to fungal penetration and the others in resistance to spread {10503}. There were significant correlations of FHB response with height and heading date {10503}. Qfhs.lfl-1BL was verified in F4:7 lines and detected in Biscay, History and Pirat {10768}. The renamed Qfhs.lfl-1BL reduced FHB severity by 42% relative to lines lacking it {10698}. This gene was also present in Biscay, History and Pirat {10698}. CS / CS(Sumai 3 7A): QFhb7AC , nearest marker Xwmc17-7A , explained 22% of phenotypic variance for Type II and 24% of phenotypic variance for Type III resistance {10798}. Chris / Frontana: In a reciprocal backcross analysis of Chris monosomics/Frontana, Frontana chromosomes 3A, 6A and 4D reduced visibly diseased kernels, kernel weight and DON content, whereas Frontana chromosomes 2A, 2B, 4B and 7A increased the same traits {10398}. Further study of the 3A, 6A and 4D reciprocal substitution lines indicated that chromosome 3A of Frontana had the largest effect on incidence, severity, spread and kernel damage, 4D less so and 6A possibly not at all {10900}. DH181(R)(Sumai 3 / HY 386 Seln.): QTL identified in 2DS, 3AS, 3BS, 3B Cent. region, 4DL, 5AS, 6BS {10213}. Dream(R) / Lynx(S): RIL population: Following inoculation with F. culmorum 4 QTL for AUDPC were identified on chromosomes 6AL (R[2] =19%), 1B (12%), 2BL (11%) and 7BS (21%). The resistance allele in 1B came from Lynx and was associated with T1BL.1RS {10260}. Dream*4 / Lynx lines were developed by selection of QTL on chromosomes 6AL, 7BS and 2BL. Lines carrying QFhs.lfl-6AL and QFhs.lfl-7BS were more resistant than lines lacking them; the 2BL QTL effect was not verified {10470}. Chokwang (R) / Clark (S): Qfhb.ksu-5DL.1 associated with Xbarc239-5D (R[2] =0.24) {10276}, Qfhb.ksu4BL.1 associated with Xbarc1096-4B (R[2] =0.13) {10276}, and Qfhs.ksu-3BS.1 marginally associated with the region of Fhb1 (R[2] =0.1) {10276}.Ernie (Res) / MO94-317 (Sus): 243 F8 RIL population. Four QTLs from Ernie detected as follows: Qfhs.umc-2B , linked to Xgwm278-2BS , R[2] = 0.04 {10456}; Qfhs.umc-3B , linked to Xgwm285-3BS , R[2] = 0.13 {10456}; Qfhs.umc-4B , linked to Xgwm495-4BL , R[2] = 0.09 {10456}. Qfhs.umc-5A , linked to Xgwm165-5A , R[2] = 0.17 {10456}. Evidence was provided to suggest the QTL acted additively {10456}. Frontana (R) / Remus (S): Major QTLs in chromosomes 3AL ( Xgwm270-3AL – Xdupw227-3A region) and 5A ( Xgwm129-5A – Xbarc-5A region) accounted for 16% and 9% of the phenotypic variation (mainly type 1 resistance) over 3 years {10174}. Frontana (MR) / Seri82 (S): F3 and F3:5 populations: QTLs were located in chromosomes 1BL (R[2] =7.9%), flanked by AFLP markers, 3AL (R[2] =7.7%), flanked by Xgwm720-3A and Xgwm121-3A , and 7AS (R[2] =7.6%), flanked by anAFLP and Xgwm233-7A {10349}. G16-92 (R) / Hussar (S): Two QTL for resistance to F. culmorum were identified on chromosome 1A (resistance from Hussar) (R[2] = 0.01) and 2B (resistance from G16-92) (R[2] = 0.14) {10588}. Glenn (R) / MN00261-4 (S): RIL population: three of 15 QTL for FHB response and heading date were stable and explained >10% of the phenotypic variation; these were located on chromosome arms 5BL, 6BS (possibly Fhb2 ) and 7AS {11568}. Grandin (S) / PI277012 (I): DH population: Two QTLs, Qfhb.rwg-5A.1 on 5AS (R[2] 0.06-0.2) and Qfhb.rwg-5A.2 on 5AL (R[2] =0.12-0.2) conferred type I and II resistance and reduced DON content {0147}. The new QTL on 5AL was closely but not completely linked with gene q which is present in PI277012 {10860}. Hobbit Sib / T. macha

7DL-7el2 {10489}. 7el2{10489}.

segregation with Xcsl107-2B . Four of 6 land varieties possessed Xcsl107-2B . A variant haplotype of Xcsl107-2B was present in AUS4930 {10013}; Xcdo36-2B – 7.5 cM – Xbcd1231-2B/XAtPPr5/Xcsl1072B/Cre1 {10013}.

2AS {0107} = 2A-2N[v] -6N[v] .

6BL {0220}, on basis of linkage with Xbcd1-6B and Xcdo347-6B {220}.

Qcre.src-1B was located to the Xwmc719-1B (R[2] =12%) – Xgwm140-1B (R[2] =12%) region in Trident/Molineux {10343}. For review {11309}.

RMG6 . TraesCS1D02G029900 .

Rmg6 was present in 77% of surveyed genotypes 11470}. Xwmc432-1D – RMG9 – 5.0 cM – RMG6 – Xwmc222-1D {11504}. A second gene in chromosome 1D designated Rwt4 {0302} ( TraesCS1D02G058900 {11632}) (was present in CS and Norin 4.

Among PM4 alleles Pm4a, Pm4b and Pm4d conferred resistance to both MoT and Bgt ; Pm5f conferred resistance to MoT but not Bgt , and Pm4f was ineffective against both pathogens {11735}.

RmgGR119 confers resistance to the wheat form of the pathogen and its response is enhanced in combination with Rmg8 {11263}. Near-isogenic lines with the T2A-2NS translocation from Ae. ventricosa displayed reduced levels of spike blast, but there was little effect on seedling leaf blast response {11265}:

v2 Chinese Spring Rmg6 {11504}.

RmgTd(t) was detected with a white culture of an Avena pathogen isolate backcrossed to a wheat isolate. A virulence to RmgTd(t) was completely associated with white color of the pathgen isolate {10949}. The white color appeared as a mutant variant during backcrossing. The wheat blast pathogen became established on wheat cultivar Anahuac ( rmg1 rmg6 ) in Brazil in the mid-1980s. It was initially avirulent on cultivars such as IAC-5 with Rmg6 but later acquired virulence allowing it to attack most wheat genotypes {11470}.

Allan et al. {019} considered that H3 and H4 might be allelic. Also suggested by Shands and Cartwright {1317}. Linkage of 10.5 +/- 2% involving H3 and Pm3a in PI 468960 was attributed to a chromosome 1A/5A translocation {1437}.

Recessive. 1AS {11634}. H4 confered resistance to race A, but not to race B.

H7. 6AS {11511}; 5D {026}.

Based on the location of H9 on chromosome 1AS, H15 may also be located on chromosome 1AS {10231}.

2R#2L {389}.

6BL-6R#1L {389}

H26 is very close to H32 {10846}. H27

Halotype analysis was used to postulate Ae. tauschii -derived genes H13, H22, H23, H26 and H32 in a set of synthetic wheat lines {10983}. H35 in chromosome arm 3BS and H36 in chromosome arm 7AS were named for one major and one minor QTL in common wheat line SD06165 {11512}. Temporary designations:

A recombination value of 12.0% between leaf-rust reaction {possibly Lr10 } and Hessian-fly reaction in Selection 5240 was reported {018}.

Duster (R) / Billings: DH population: QHf.osu.1A.2 (Syn. QHf.osu-1A[d] ), R[2] = 0.88, delimited to a 2.7 cM region flanked by GBS07851 and GBS10205 {11324}. This was a distinct locus 11.2 cM proximal to QHf.osu.1A . Jagger (S) / 2174 9 (R): RIL population: QHf.osu-1A (Syn. Qhf.osu[74] (R[2] = 0.70) and QHf.osu-2A (R[2] = 0.18) {11325}. The QTL in chromosome 1A appeared to be co-linear with several previously named H genes in tetraploid wheat; the gene in 2A was in repulsion with the 2N segment present in Jagger {11325}.

{10486}; May be the same as CreY (see reaction to Heterodera avenae ) on chromosome 3S[V] from Ae. variabilis translocated to 3BL {10800}.

6D, {10105} (according to {10556} this location is not correct.

{0187}; Israel 493 {11446}; Longbow {11446}; Olaf {11446}; Sears' Synthetic {0186}; Senat {11446}; Shafir {0}; Veranopolis {11446}; Vivant {0187}.

See {11332, 11361} for reviews. Temporary designation

QTL Four QTLs for resistance to Mycosphaerella graminicola were identified in replicated field experiments in a double haploid population from Savannah (susceptible)/Senat (resistant) . Senat contributed all the alleles providing resistance {10067}. QStb.riso-2B was mapped on chromosome arm 2BL linked to SSR marker Xwmc175-2B (LOD>5, R[2] >17%) {10067}. QStb.riso-3A.2 was mapped on chromosome arm 3AS linked to SSR markers Xwmc489-3A, Xwmc3883A and Xwmc505-3A (LOD >4, R[2 ] >18%). Also detected at the seedling stage {10067}. Xgwm369-3A is present on chromosome arm 3AS {0187}. A resistance gene from Senat located at or near the STB6 was mapped 5 cM from Xgwm369-3A on chromosome arm 3AS {10067}. QStb.riso-6B was mapped on the centromeric region between SSR markers Xwmc494-6B and Xwmc3416B (LOD >16, R[2 ] >68%). Also detected at the seedling stage {10067}. QStb.riso-7B was mapped on chromosome 7B close to SSR marker Xwmc517-7B (LOD>4, R[2 ] >11%) {10067}. ITMI Population: Three QTL, QStb.ipk-1DS, QStb.ipk-2DS and QStb.ipk-6DS conferred seedling-stage resistance to 2 isolates, whereas 2 QTL QStb.ipk-3DL and QStb.ipk-7BL conferred separate adult-stage resistances to each isolate {10151}. A weak QTL, QStb.psr-7D.1 , giving partial resistance to Portuguese isolate IPO92006, was detected in the Xcdo475b-7B - Xswm5-7B region in chromosome 7DS {10341}. Apache / Balance: Analyses with a panel of M. graminicola cultures identified QTLs on chromosomes 1BS (Apache, considered to be Stb11 ), 3AS (Balance, considered to be Stb6 ), 6DS (Balance, named as Stb18 ), 7DS (Apache, considered to be Stb4 ) and 7DL (Apache) {10827}. Florett / Biscay (S): RIL population: two QTLs for APR were located on chromosomes 3B and 6D {10901}. Solitar (R) / Mazurka (S): DH population: Resistance under field conditions was associated with QTL on chromosomes 5A, 6D and 7D which accounted for 20% of the genotypic variation; all three were derived from Solitar, but there was no evidence that Stb6 and Stb11 , also present in Solitar, were involved {10984}. Spelt HRTI1410 (R) / three wheat parents: 135 DH lines: mapped using SNP polymorphisms common to all three S parents: four QTL identified on chromosome 5AL (74.2 – 82.4 cM; r[2] = 0.18); 4B (52.9 – 56.9 cm, r[2] = 0.09) contributed by the susceptible parents; and 7B.1 (41.2 – 57.0 cM, r[2] = 0.09), and 7B.2 (58.2 – 67.4 cM, r[2] = 0.15) contributed by the susceptible parents {11430}. Steele-ND (R) / ND735 (S): RIL population: A consistent QTL (R[2] =0.1) for seedling resistance flanked by DArT markers XwPt-7101 and X377410 was mapped to chromosome 5BL in the region of Stb1 {10992}. Two other QTLs on chromosomes 1D and 7A were detected in single experiments {10992}. Tuareg / Biscay (S): RIL population: two QTLs for APR were located on chromosomes 4B and 6B {10901}. For a review of qualitative and quantitative resistance {11439}.

QTL A QTL analysis of SNB response in the ITMI population found significant effects associated with chromosome 1B (probably Snn1 ) and 4BL, with an interactive effect involving the 1BS region and a marker on chromosome 2B {10009}. An additional QTL on 7BL was effective at a later stage of disease development {10009}. Arina / Forno: RIL population {10065}. Two QTLs for glume blotch resistance under natural infection were identified on chromosomes 3BS and 4BL in. QSng.sfr-3BL was associated with marker Xgwm3893B and explained 31.2% of the variation with resistance contributed by Arina {10065}. The 4BL QTL, QSng.sfr-4BL , was associated with Xgwm251-4B and explained 19.1% of the variation. Resistance was contributed by Forno {10065}. A QTL on 5BL, QSng.sfr-5BL , overlapped with QTLs for plant height and heading time {10065}. QSng.sfr-3BS peaked 0.6 cm proximal to Xsun2-3B {10465}. Association mapping involving 44 modern European cultivars indicated that the association was retained in a significant proportion of genotypes {10465}. Br34 / Grandin: Three QTLs with resistance effects from BR34; Qsnb.fcu-5BL.1 ( Tsn1 ), R[2] = 0.63, Qsnb.fcu5BL.2, R[2] = 0.06, and Qsnb.fcu-1BS (vicinity of Snn1 ), R[2] = 0.10 {10458}. QTL analysis of the RIL population with Culture Sn6 revealed four QTLs, Qsnb.fcu-2DS (R[2] = 0.3 - 0.49) associated with Snn2 , Qsnb.fcu-5BL (R[2] = 0.14 - 0.2) associated with Tsn1, Qsnb.fcu-5AL (R[2] = 0 - 0.13) associated with Xfcp13-5A , and Qsnb.fcu-1BS (R[2] = 0 - 0.11) associated with Xgdm125-1BS {10507}. Forno (S) / Oberkulmer spelt (R): Among 204 RILs leaf and glume response were genetically different but correlated (R[2] =0.52). Ten QTLs for glume blotch (SNG) resistance were detected, 6 from Forno. A major QTL (R[2] =35.8%) was associated with q. Eleven QTLs (4 from Forno) affected leaf blotch; 3 of these (chromosome 3D, 4B and 7B) with R[2] >13% were considered potential candidates for MAS {10250}. HRWSN125 (R) / WAWHT2074 (S): Constant detection of QSnl.daw-2DL for flag leaf resistance, and QSng.daw-4BL for glume resistance over two years {10584}. ITMI population: A major QTL, coinciding with Snn1 , was located in chromosome 1BS (R[2] = 0.58, 5 days after inoculation), minor QTL were found in 3AS, 3DL, 4AL, 4BL, 5DL, 6AL and 7BL {10009}. P91193D1 (partially resistant) / P92201D5 (partially resistant) RIL populations were tested in Indiana and Western Australia for glume resistance. Two QTL were identified: Qng.pur-2DL.1 from P91193D1 (R[2] = 12.3 in Indiana and 38.1% in WA, respectively; Xgwm526.1-2D - Xcfd50.2-2D ) and QSng.pur2DL.2 from P99201D5 (R[2] = 6.9% and 11.2%, respectively; Xcfd50.3-2D - wPT9848 ) {10471}. Liwilla / Begra: DH population: Four QTLs, on chromosomes 2B (proximal part of long arm), 3B (distal part of short arm), 5B and 5D. A longer incubation period and lower disease intensity were contributed by Liwilla {10045}. A QTL, QSnl.ihar-6AL , identified in DH lines of Alba (R) / Begra (S) accounted for 36% of the phenotypic variance in disease severity and 14% of the variance in incubation period {10143}. Salamouni/Katepwa: RIL population: Two QTLs. QSnb.fcu-1A ( Snn4 ) (R[2] =0.24) and QSnb.fcu-7A (R[2] =0.16) were associated with SNB response to isolate Sn99CH 1A7a {10867}. ## Tetraploid wheat Langdon / Langdon ( T. turgidum ssp. dicoccoides Israel-A 5B): QSnb.ndsu-5B located 8.3 cM proximal to tsn1 for tan spot resistance; R[2] = 0.38 {10597}. A summary of QTL analyses is provided in {10726}.